Maturation Phenomenon in Cerebral Ischemia IV Apoptosis and/or Necrosis, Neuronal Recovery vs. Death, and Protection Against Infarction. - Taschenbuch

EAN (ISBN-13): 9783540411079
ISBN (ISBN-10): 3540411070
Taschenbuch
Erscheinungsjahr: 2001
Herausgeber: Springer Berlin

Buch in der Datenbank seit 2007-06-05T10:26:55+02:00 (Berlin)
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ISBN/EAN: 3540411070

ISBN - alternative Schreibweisen:
3-540-41107-0, 978-3-540-41107-9
Alternative Schreibweisen und verwandte Suchbegriffe:
Autor des Buches: bazan, ito, marcheselli, marchese
Titel des Buches: recovery, protection, death, and, the cerebral, matura

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Daten vom Verlag:

Autor/in: N.G. . Bazan; U. Ito; V.L. Marcheselli; T. Kuroiwa; I. Klatzo
Titel: Maturation Phenomenon in Cerebral Ischemia IV - Apoptosis and/or Necrosis, Neuronal Recovery vs. Death, and Protection Against Infarction
Verlag: Springer; Springer Berlin
284 Seiten
Erscheinungsjahr: 2001-03-27
Berlin; Heidelberg; DE
Gedruckt / Hergestellt in Deutschland.
Gewicht: 0,461 kg
Sprache: Englisch
106,99 € (DE)
109,99 € (AT)
118,00 CHF (CH)
POD
XVI, 284 p. 54 illus.

BC; Neurology; Hardcover, Softcover / Medizin/Klinische Fächer; Neurologie und klinische Neurophysiologie; Verstehen; Cerebral ischemia; apoptosis; cerebral infarction; delayed neuronal death; forebrain; neuronal necrosis; neurons; Neurosurgery; Internal Medicine; Emergency Medicine; Anesthesiology; Neurology; Neurosurgery; Internal Medicine; Emergency Medicine; Anesthesiology; Neurochirurgie; Klinische und Innere Medizin; Notfallmedizin; Anästhesiologie; EA

I The Role of Genetic Expression and Apoptosis.- A Role for the GTPase Pathway in Neuronal Damage After Cerebral Ischemia: the Impact of the DNA Array Technique on Stroke Research.- Induction of Hypoxia-Inducible Factor-1 (HIF-1) in Adult and Neonatal Rat Brain: Possible Relationship to Hypoxia-Induced Tolerance to Ischemia.- Characterization of the eIF2-Associated Protein p67 During Brain Ischemia and Reperfusion.- Is DNA Methylation Deleterious in Cerebral Ischemia?.- Distinct Ischemic Effects on HSC70, HSP72, and c-fos Expression in Young and Adult Gerbils.- Bcl-w Expression and Localization in Brain Ischemia.- II Factors Modulating Neuronal Plasticity and Course of Maturation Phenomenon in Cerebral Ischemia (Metabolic and Inflammatory Factors).- Thrombosis After Ischemic Stroke-Platelet Aggregation.- The Role of the Immunophilin FKBP12 in Cerebral Ischemia.- Localization of Macrophage Migration Inhibitory Factor in the Brain Following Focal Cerebral Ischemia in the Rat.- Environmental Influence on Neuronal and Dendritic Spine Plasticity After Permanent Focal Brain Ischemia.- Relationship Between DNA Fragmentation, Energy State, and Protein Synthesis After Transient Focal Cerebral Ischemia in Mice.- Changes in Postsynaptic Densities After Brain Ischemia.- Functional Plasticity of the Brain After Ischemic Injury Assessed by Near Infrared Topography.- III Factors and Mechanism Enhancing Susceptibility or Tolerance (Growth Factors).- TNF-? and Ceramide as Mediators of Neuronal Tolerance to Brain Ischemia.- Upregulation of Transforming Growth Factor-Beta 1 Expression in Rat Hippocampal Neurons After Transient Forebrain Ischemia and After ?2-Adrenoceptor Stimulation.- Bcl-2-Protein Is Upregulated After Chemical Induction of Ischemic Tolerance Using 3-Nitropropionic Acid in Rats.- Time Course of Postischemic Stroke Symptoms and Delayed Infarction After Transient Cerebral Ischemia in Gerbils: Effect of Chemical Preconditioning Using 3-Nitropropionic Acid.- Cyclic AMP Response Element Binding Protein Phosphorylation May Be Closely Associated with Neuroprotective Mechanisms After Focal Ischemia in Rat Brain.- Recent Evidence on the Neuroprotective Effects of Pituitary Adenylate Cyclase-Activating Polypeptide.- Evolution of Microcirculatory Derangement in Focal Ischemic Penumbra Following Pial Arteriolar Occlusion as Observed by the Novel Photoelectric Method.- IV Ischemic Infarction: Threshold, Experimental and Clinical Dynamics and Therapeutic Design for Prevention and Reduction of Intensity.- Delayed Maturation of Cortical Infarction: Role of Caspases and NF-AKB-Mediated Transcription.- Cellular Components with Adaptive Responses Contributing to Expansion and Repair Process of Ischemic Brain Damages Caused by Major Vessel Occlusion.- Combination Drug Therapy and Mild Hypothermia: Comparison with Neurosurgical Standard Regimen in a Rat Model of Reversible Focal Cerebral Ischemia.- Thrombolysis in Acute Stroke.- V Mitochondrial Role in Ischemic Cell Death.- Cytochrome c Is Released from the Mitochondria of Vulnerable Hippocampal CA1 Neurons in Rats After Transient Global Cerebral Ischemia.- Formation of 4-Hydroxy-2-Nonenal-Modified Proteins in the Rat Brain Following Transient Global Ischemia Induced by Cardiac Arrest and Resuscitation.- Mitochondrial Dysfunction and Maturation of Brain Damage After Transient Ischemia.- Hypoxia-Induced DNA Damage in the Rat Brain.- Ultrastructure and Morphometry of Astroglial Mitochondria Following Temporary Ischemia.- VI Special Lecture.- Translating Experimental Stroke Research into the Clinical Arena.
Fourth International Workshop, October 30-November 3, 1999, New Orleans, Louisiana, USA;